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Research models reveal that senescent cells accumulate with aging and in response to various stressors, secreting inflammatory factors known as the senescence-associated secretory phenotype (SASP) that promotes chronic inflammation and can induce senescence in normal cells

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These findings suggest that ionophore-mediated Cu delivery to the intracellular compartment might be a promising therapeutic strategy for a subset of tumors, and additional Cu–ionophore complexes that target cuproptosis warrant further development