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However, because excretion of vitamin B12 in the stool is proportional to B12 body stores, B12 defi­ciency develops more slowly in persons han in persons with no intrinsic factor or with malabsorption 22b.1.4 Deficiency of vitamin B12 in humans The common consequence of B12 defi­ciency is a cellular deficit in one of the coenzyme forms of B12 which affects cellular meta­bolism, methylation and mitochondrial meta­bolism: such deficits may pass from a stage of subclinical deficiency through to severe, clinical forms

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The voltage-gated Cl − channels are predominantly found in astrocytes, oligodendrocytes, pyramidal and non-pyramidal neurons in the brain