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While iron is essential for physiological processes such as hemoglobin synthesis and redox reactions, dysregulation of cellular iron homeostasis—mediated by transferrin receptor 1 (TFR1) for uptake, ferritin heavy/light chains (FTH1/FTL) for storage, and ferroportin 1 (FPN1) for export—results in labile iron pool expansion

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Mitochondria (the “power plants” inside cells) don’t work well in neuropathy, which lowers energy (ATP) and produces harmful molecules called free radicals