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Second, accelerated alcohol metabolism by higher levels of CYP2E1 puts liver cells in metabolic peril, because more CYP2E1 not only produces more acetaldehyde, but the induced enzyme also generates greater amounts of various other reactive oxygen species (ROS), including hydroxyethyl radicals (i.e., free-radical forms of ethanol), superoxide anions (O 2 − ) and hydroxyl radicals (·OH)