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Finally, the KD increases fatty acid oxidation by raising polyunsaturated fatty acid (PUFA) levels, which can reduce neuronal excitability and mitigate seizures by blocking voltage-activated sodium and Ca++ channels, activating K2P channels, and enhancing Na+/K+ ATPase pump activity [40]

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DSAPGS motif interacts with β-TrCP, which serves as a substrate receptor for the S-phase kinase-associated protein 1- Cullin 1- RING box protein-1/regulator of cullins-1 (Skp1–Cul1–Rbx1/Roc1) ubiquitin ligase complex