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Chronic Doxycycline treatment functionally confers a mitochondrial-deficient metabolic phenotype, actively suppressing CSC activity Previous studies have shown that human cancer cells lacking mt-DNA [called rho(0) cells] have largely lost their ability to undergo mitochondrial OXPHOS and they fail to initiate tumors in vivo , as determined by using pre-clinical animal models to assess tumorigenicity [3]

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Effects of supranutritional selenium nanoparticles on immune and antioxidant capacity in Sprague-Dawley rats

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