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It has been indicated in a study that genistein could induce apoptosis in HT-29 and LoVo human colon cancer by upregulating Bax and downregulating the NF-κB pathway, thus supporting the probable application of genistein in clinical application for treating colon cancer patients [76]

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Excessive intracellular Fe 2+ will generate excessive ROS through the Fenton reaction, leading to a continuous accumulation of intracellular ROS and triggering ferroptosis (Zhao et al., 2023)