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Inflammatory and Oxidative Stress Research GHK-Cu has also been explored in research models examining inflammatory signaling and oxidative stress regulation, including: Cytokine Expression Studies: Evaluation of inflammatory mediator patterns in dermal and connective tissue systems Antioxidant Enzyme Regulation: Investigation of superoxide dismutase and related copper-dependent enzymes Reactive Oxygen Species Modulation: Research into redox balance within cellular environments Tissue Remodeling Pathways: Examination of signaling systems balancing inflammation and repair processes Laboratory protocols assess oxidative biomarkers, inflammatory mediators, and matrix composition using biochemical and imaging-based techniques

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Conclusion: These findings suggest that Fasn inhibition may offer a promising therapeutic strategy for hypertension-induced ED by alleviating oxidative stress and suppressing NLRP3 inflammasome-dependent endothelial cell pyroptosis via activation of the Nrf2/HO-1 pathway