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Thus, some G-quadruplex-targeting RBPs regulate RAN translation and G 4 C 2 repeat-induced toxicity by binding to and possibly by modulating the G-quadruplex structure of G 4 C 2 repeat RNA

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Another mechanism of GSH protection is reverse post-translational modifications of proteins by S-glutathionylation/deglutathionylation), which is common for the Krebs cycle and the electron transport chain proteins catalyzed by glutathione-S-transferase and glutaredoxins (Marí et al., 2020)