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It has been established that PRMT1 is recruited by DNA-PK (DNA-dependent protein kinase) to chromatin where it catalyzes the methylation of H4R3 and contributes to the stimulation of genes involved in the senescence-associated secretory phenotype (SASP), in turn increases tumor cells' resistance to cisplatin by shielding them from DNA damage [88]

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This finding forms the rational basis for the use of combinations of alkylating agents in high-dose chemotherapy regimens