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In murine models, HBOT has been shown to potentiate this signalling pathway by upregulating the expression of SIRT1, PGC-1α, TFAM, and voltage-dependent anion-selective channel proteins, confirming its ability to induce mitochondrial biogenesis and enhance mitochondrial functionality [72]

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The donor alveolar macrophages appear to play a critical role in the initiation of lung damage upon reperfusion, by secreting inflammatory cytokines, resulting in the secondary recruitment of recipient activated neutrophils, with ensuing inflammation, epithelial and endothelial damage leading to reperfusion lung edema (397)