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Bottom line: BPC-157 has a coherent and interesting proposed mechanism — centered on angiogenesis through VEGFR2–Akt–eNOS signaling, supported by effects on the FAK–paxillin pathway, the nitric oxide system, and growth factor receptors — but this mechanism is demonstrated in animals, not humans

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Scaling, biological fouling, or fill media degradation can erode thermal performance and increase energy consumption