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Increased production of superoxide radicals indiscriminately induce oxidative damage via several mechanisms such as initiation of lipid peroxidation, inactivation of antioxidant enzymes, attacks phospholipids, proteins and DNA, and damages protein sulfhydryl oxidation leading to neuronal loss and reactive gliosis resulting in impairments in neuronal function and cognition, even cell death (Peluffo et al., 2005, Zhang et al., 2022)
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The results of the serum metabolome in this experiment showed that the levels of PC, PS, and PE were significantly decreased in the FI group, whereas their levels were significantly up-regulated after the intervention of glutathione administration, suggesting that glutathione alleviates glycerophospholipid metabolism disorders to a certain extent, and thus hepatic lipid metabolism disorders
