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IGF-1R is composed of two extracellular α-subunits that are activated upon IGF-1 binding and two β-subunits that have intracellular tyrosine kinase domains that are phosphorylated by IGF-1

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Overexpression of miR-155 decreases C/EBPβ levels, thereby reducing the accumulation of fatty acids within lipid droplets in the liver [322]