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Glutathione in cancer progression and chemoresistance: an update - ScienceDirect

FIGURE 1 The capability of INH to cross the cell membranes simply by passive diffusion and selective action against Mtb accords with its high therapeutic potential (Bardou et al., 1998)

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Treatment with a thiol ester compound (gamma-glutamylcysteinyl ethyl esther) or exogenous GSH while examining cellular models of diminished GSH levels in concert with Complex I inhibitors MPP + , MPTP, or DHBT-1, restores mitochondrial Complex I activity, inhibits cell loss and protects against cell and striatal DA loss [73, 75, 82]