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Although the impairment of cysteine uptake by EAAC1 gene deletion increases free zinc levels in neurons, we demonstrated that NAC treatment reduced neuronal free zinc levels in EAAC1-knockout mice [13,72,73,74]

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Taken together, the above-mentioned data suggest that GSH might serve as an important wound signal that triggers wound-induced systemic [Ca 2+ ] cyt transmission, activates JA biosynthesis, and regulates plant defense responses