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HTyr treatment decreased both, posthypoxic ROS and NO levels and consequently, hypoxia-inducible factor-1 (HIF-1) accumulation, and promoted the expression of angiogenic proteins, suggesting that HTyr activates an adaptive response to hypoxia in a HIF-1α-independent pathway that could be ascribed to the up-regulation of estrogen-related receptor α

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J., Faull, R